Biliary Colic Prevention and Treatment


Preventive measures are addressed to patients with gallstones, those who have had this issue or who show biliary dyspeptic manifestation.

gallbladderThe goal is to improve biliary dyskinesia, address the causes (biliary stasis, biliary infections, obesity, constipation, use of oral contraceptives) that favour lithogenesis (formation of gallstones) and the neuro-vegetative factors (stress, emotions, and conflicting states).

Diet: the recommendations are orientative only, each patient requiring an individualized regimen based on their specific tolerance.


  • split meals (4-5 per day) in moderation quantities;
  • lying idle (half hour) after meals;
  • food not too hot and well chewed;
  • reduction or quitting smoking.

Prohibited foods:

  • fat milk, sour cream, cream, cheese products;
  • fat soups, pulses (beans, peas, lentils);
  • fat pork, goose, duck, smoked cured meats, sausages, bacon, ham fat, organs (brain, kidney, liver), fatty fish (carp, catfish);
  • eggs or omelette with bacon;
  • food with sauce, goulash, paprika, spices (pepper, mustard, olives);
  • fruit: walnuts, hazelnuts, peanuts, almonds;
  • many cakes made with eggs, cream, fatty creams, ice cream;
  • black coffee, chocolate, cocoa, alcoholic beverages (beer, brandy, liqueur).

Foods without fat are generally allowed, salty foods with moderation, pasta, green vegetables cooked with butter or oil (sautéing or purées), buttermilk, yogurt, skim milk, cottage cheese fresh poultry poor (boiled or barbecue) without sauces, veal, lamb, little snacks, salted eggs, cheese or apple pie, raw fruits or baked, stewed – are allowed.

Besides the presented diet, it may also be recommended medication according to the needs, choleretic and kinetic (metoclopramide), spasmolytic (lizadon, Scobutil) and sedatives (hydroxyzine, Extraveral).

Treatment of biliary colic: aims to relieve pain, nausea, vomiting and anxiety.

Bed rest is recommended during the painful phase.

  1. Medical treatment is addressed to uncomplicated biliary colic. It consists of analgesic and antispasmodic medication (Algocalmin, Scobutil, Papaverine). Analgesics and antispasmodic preparations may be associated, their effects empowering each other. If pain does not respond to antispasmodics further investigations are required depending on the instructions of the doctor, possibly hospitalization.

Antiemetic medication: Emetiral, Torecan, sedative medication: hydroxyzine, napoton, meprobamate.

Generally, uncomplicated biliary colic is resolved by this treatment. Investigating the causes that caused it and the frequency, the severity, the presence of another disease, allows establishing whether surgical treatment is indicated.

  1. When is surgical treatment indicated?

Surgical treatment of biliary colic aims to solve the problem of complicated biliary colic:

  • gallbladder hydrops (mechanical obstruction of the bile duct – the cystic duct);
  • acute cholecystitis (inflammation + hydrops of the gallbladder wall);
  • piocolecist (suppurative inflammation of the gallbladder + hydrops);
  • gangrenous cholecystitis (gallbladder microabscesses in the walls);
  • biliary peritonitis (gallbladder perforation cavity peritoneal);
  • biliary fistulas (perforated in a neighbouring organ);
  • bile duct stones (migrating calculi from the gallbladder bile => jaundice, fever);

These complications may be life-threatening and emergency surgical intervention is indicated regardless of the age or the associated disorders that cause increased operational risk.

Because of the risk of unpredictable complications, surgical treatment is recommended for all patients with symptomatic gallstones.

Of those, the most vulnerable to develop complications are:

  • patients who have experienced more than 2 episodes in the last 3 months;
  • patients who have a history of complicated colic (hydrops, acute cholecystitis, bile duct stones) that were resolved by conservative treatment;
  • patients who presented with acute pancreatitis with complicated biliary colic.
  • patients presenting other diseased which are aggravated by the biliary colic and stones (diabetes, ischemic heart disease, chronic pancreatitis, chronic hepatitis).

In these cases, surgical indication will be individualized taking into account the increased risk of developing complications. Surgery may be indicated for prophylaxis even in patients with few or no symptoms.

The surgery consists of cholecystectomy (surgical removal of the gallbladder). This can be done in the conservatory method or laparoscopic method.

Classic cholecystectomy is performed under general anaesthesia, with subhepatic or median approach. It provides increased surgical comfort, any other complications can be resolved (peritonitis, bile duct stones, fistulas). The patient needs 8-10 days of hospitalization.

Laparoscopic cholecystectomy is the modern alternative. It is aimed especially to the uncomplicated cases. Advantages: the recovery is much faster (1-2 days of hospitalization) and for cosmetic reasons.

Posted in Digestive system

Precancerous conditions that portend cancer

Precancerous conditions

The vast majority of cancers are preceded by a precancerous condition (90% of cancers), but the process is often very long. The malignant evolution of precancerous conditions is not mandatory nor can specify an exact time for a malignant transformation. The duration of the evolution is variable from an organ to another, from a few years to tens of years.

Precancerous conditionsFor these lesions there are different names used: precursor states, border, precancerous.

Since about 90% of cancers appear as a result of the development of precancerous conditions, it is extremely important to recognize them and the associated etiological factors and to remove them.

Once discovered, the precancerous lesion should be treated accordingly, given the fact that it is considered a major risk factor for cancer.

The recognition and correct treatment of premalignant conditions

– Allows clinical cancer prevention,

– The costs involved are much lower than for invasive cancers,

– Allows healing the patients.

WHO Classification divides the precancerous conditions into 2 groups: precancerous conditions and precancerous lesions.

Precancerous conditions: are biological statuses, histological and clinical, with a high tendency not to develop into cancer, but whose exogenous carcinogen factors are not known.

They are represented by:

a) Chromosomal or genetic anomalies;

b) Hereditary disorders with risk of cancer;

c) Various dystrophies and benign diseases (adenomas, polyps).

brca2Oncogenes can cause certain medical conditions and biological precursors or some cancers: the direct transmission of BRCA2 gene cause gynecomastia with unilateral channeling in men, followed by malignant swelling.

Chromosomal abnormalities: trisomy 21, Klinefelter syndrome (XXY syndrome), the syndrome caused by the deletion of chromosome 13. Trisomy 21 is associated with a higher risk of developing acute leukemia and solid tumors (10-20 x). Chromosome 13 deletion syndrome is associated with high risk of bilateral retinoblastoma.

Polyposis syndromes of hereditary colon (Turcot, Gardner) are associated with a high risk of malignancy.

Peutz-Jeghers syndromePeutz-Jeghers syndrome predisposes to cancer of the colon, breast cancer with the phenotypic markers: hyper pigmented spots in the oral region, oral mucosa, and extremities.

Xeroderma pigmentosum (rare inherited disorder characterized by defective transmitted recessive DNA repair) causes multiple skin cancers in people exposed to UV radiation during childhood. Originally, there are some blisters at birth, small erythematous bumps, then they become hyper or hypo pigmented. At 3-4 years, benign cutaneous tumors appear, which turn into melanomas, cancers of the eyelids, cancer of the cornea associated with mental retardation, epilepsy.

Fanconi Anemia (hereditary transmission, with phenotypic markers like anemia, microcephaly, low height, horseshoe kidney, skin pigmentation, eye abnormalities) predisposes to acute myeloid leukemia, cancers of the mucocutaneous junctions, liver cancer.

Vincent Plummer syndrome – young women with iron deficiency anemia due to  esophageal dysphagia caused by a diaphragm. This disease requires endoscopic surveillance because it is associated with an increased risk of esophageal cancer.

Ataxia syndrome (hereditary transmission, consisting of: progressive cerebellar ataxia, ocular and cutaneous teleangectasy, immune deficiency) predisposes to the appearance of lymphomas.

RecklinghausenRecklinghausen Neurofibromatosis (autosomal dominant transmission) – neurofibromas, brown pigmented spots, molluscum – frequently evolves into sarcomas and predisposes to the occurrence of cerebral gliomas.

Multiple hereditary cancers: Li-Fraumeni syndrome (defect in the p53) is associated with an increased risk of breast or colon cancer, brain cancer, sarcoma, leukemia. Cowden disease can determine, in addition to the skin hamartomas, breast cancer in 30-50% of the cases or thyroid cancer.

Crohn’s disease shows an increased risk for colon cancer.

Chronic atrophic gastritis shows risk of malignancy.

Testicular dysgenesis associated with cryptorchidism is malign in 70% of the cases. Cryptorchidism has a major risk of malignant transformation. Orhidopexia does not prevent testicular cancer if performed late.

Senile keratosis – commonly associated with cutanate cancers – baldness, skin lesions, superficial brown hyperkeratotic plaques. A particular form is the cutaneous horn, located on the lower lip.

Scleroatrophic lichen or vulvar kraurozis affects women over 50 years, consists of vulvar itching, burning on urination, dyspareunia (pain during intercourse), discoloration of the vulva that has a white or yellowish color. Can develop vulvar malignancies requiring supervision of the lesion, and in case of spotting, biopsy is required.

2. Precursor or precancerous lesions: appear as a result of known external or internal risk factors . The primary lesions are diffuse, multifocal and involve altering the genetic material of cells.

From a histological point of view, precursor lesions are classified into different degrees of severity: metaplasia → dysplasia → hyperplasia → carcinoma in situ.

Hyperplasia is characterized by the excessive production of a certain type of tissue histologically normal. It may be typical or atypical.

Metaplasia is the transformation of morphological and functional differentiated tissue into another differentiated tissue: epithelial squamous metaplasia in the urothelium with the development of squamous-type tumors in the bladder; cylindrical epithelium metaplasia in squamous epithelium, apocrine metaplasia of the mammary gland epithelium.

Dysplasia consists of both architectural and cytological abnormalities that occur in adult tissues.

Dysplastic lesions may remain a long time in the state of reversibility, but at that time they may most easily become malignant.

There are three degrees of dysplasia described: mild, moderate and severe (carcinoma in situ).

Carcinoma in situ is considered by some authors a precursor lesion, while others consider it an early cancer that has 100% chance of healing if properly treated, corresponding to stage 0.

The following are some of the precancerous lesions exemplified:

The presence of certain benign proliferation in the mammary gland is associated with an increased risk of developing invasive carcinomas. These lesions are often multifocal, presenting a risk of progression to invasive carcinoma of 10-30%.

This proliferation is:

Moderate and severe hyperplasia, ductal or lobular

Sclerosing adenomatosis (increases the risk of breast cancer 1.5- 2 times)

Carcinoma in situ

Carcinogenesis in breast cancer:

Normal epithelium -> atypical hyperplasia ->cancer in situ -> invasive breast cancer.

Atypical ductal or lobular hyperplasia increases the risk of breast cancer 4-5 times than the normal population.

Ductal carcinoma in situ is regarded as a real precancerous lesion preceding invasive cancer as opposed to lobular carcinoma in situ, which is more a measure of the risk of breast cancer.

Patients with a family history of breast cancer associated with personal history of atypical hyperplasia, present an increased risk of breast cancer 8 times only to those with a family history and 11 times more than the population without risk factors.

The presence of Barrett’s esophagus (adenomatosis metaplasia, severe dysplasia in the distal esophagus caused by esophageal reflux) is associated with an increased risk of esophageal cancer.

Gastric intestinal metaplasia is a common premalignant lesion and appears in 80% of the parts of gastrectomy in Japan.

Pancreatic premalignant lesions are: atypical ductal hyperplasia and ductal hyperplasia papillary and non-papillary, whose potential for malignant transformation is determined by identifying the mutated K-ras genes, using PCR. This method could be used in screening using secretions such as bile and pancreatic juice.

Leukoplakia or erythroplasia of Queyrat are precursor lesions to cancers of the upper aero-digestive tract.

Dysplastic adenomatous polyp is the main precancerous lesion in the colon.

Colorectal carcinogenesis follows this path: normal epithelium →  hyperplasia → early adenoma → intermediate adenoma→  late adenoma → cancer in situ → invasive cancer.

For preinvasive lesions of the cervix the Bethesda classification is used in the USA, which divides dysplastic epithelial abnormalities into:

Atypical changes with uncertain significance

Low-grade squamous intraepithelial lesions

Intraepithelial lesions with increased risk, comprising CIN II (moderate dysplasia), CIN III (severe dysplasia), carcinoma in situ

Endometrial hyperplasia with atypic cytology is considered severe cytologic precancerous lesion for endometrial cancer.

Precancerous skin lesions are

Bowen’s disease (intraepithelial epidermoid carcinoma of the skin and mucous membranes) which is a rare condition represented by papiliforme skin lesions with umbilical center, bluish brown, translucent, polycyclic. They have malignant transformation potential within 5-10 years.

Paget’s nipple disease, which is manifested through erythematous plaques and which may correspond to an underlying intracanalicular breast cancer or invasive cancer.

Dysplastic nerves, which can be congenital or acquired, are precursors of malignant melanoma.

Solar keratosis – skin lesions appear on the areas exposed to the sun, which are irregular, variable in color, covered with scaly keratotic skin and have a high risk of malignancy.

The precancerous lesions should be properly diagnosed and treated as they are considered a major risk factor for cancer. The development of precancerous conditions varies from several years to several decades. This range is important because of the possibility to identify and treat the lesions.

Posted in Cancer, Oncology

People at high risk of developing cervical, endometrial or breast cancer

breast cancer

breast cancerRisk factors are those that increase the likelihood that people exposed to them develop cancer. They can be dietary factors, professional, behavioral, endocrine, genetic, or drug related.

The identification of high-risk population groups based on these risk factors allows doing a cancer prophylaxis through both periodic clinical and laboratory controls, screening and through the treatment of precancerous conditions identified.

The breast cancer risk group

Age over 45 years; a breast lump is more common: an adenoma before 30 years or fibrocystic mastitis or cancer between 35 and menopause, cancer almost always at less than 2 years of menopause.

Hormonal factors: early puberty, late menopause as an expression of prolonged ovarian activity; the birth of the first child when older than 25 years, lack of breastfeeding, celibacy, nulliparity – are risk factors for breast cancer;

Familial predisposition is a major risk factor in women with first and second degree relatives who developed breast cancer (mother, sister, aunt).

Standard of living conditions predisposing to disease.

Diet high in fat, obesity.

A history of fibrocystic mastitis, ductal cancer in situ and lobular cancer in situ.

The cervical cancer risk group

Age – maximum frequency between 40-50 years, carcinoma in situ showing 10 years earlier;

There is a family predisposition for this cancer;

Lifestyle seems to have a role in the development of this cancer, which is found in women with early sexual activity and with multiple partners. It occurs frequently in prostitutes and almost never in virgins. It is more common in women with low socioeconomic level and from less developed countries.

Personal medical history is represented by common genital infections of parasitic and viral infections more often untreated: Trichomonas vaginalis, papillomavirus type A 16, 18, herpes virus type II. The presence of severe dysplasia (CIN III) can be identified in patients who will develop cancer if these lesions are not treated properly. The identification of dysplastic lesions, supervision and treatment are mandatory.

Risk group for cancer of the endometrium

Older age, most often after menopause, 60-70 years;

Hormonal factors are certainly involved in cancers of the endometrium because there of their presence of women in the following categories:

a) nulliparous

b) Having Stein-Leventhal syndrome

c) Prolonged administration of estrogen in postmenopausal women without progestin association

d) Early menarche, late menopause

Medical history:

a) Adenomatous hyperplasia of the endometrium which may be followed by the development of a carcinoma in situ and invasive cancer afterwards.

b) The triad of diabetes, hypertension, obesity is frequently observed in women who develop cancer of the endometrium.

Posted in Cancer

Migraine – Symptoms, Diagnosis, Treatment


migraineA migraine is defined as a paroxysmal headache, recurrent, unilateral, associated with ocular phenomena and autonomic disorders, which start in adolescence and in most cases, can have a family character. It affects 7-10% of the adult population. The onset is most often between late teen years and the 40s. Women are 3 times more affected.

Clinical manifestations of migraine: a succession of four distinct phases.

1.Prodromal: is present in up to 50% of cases, in the form of some symptoms with insidious onset, slow progression, lasting up to 24 hours before the crisis.

– Feeling irritable

– State of hyper reactivity

– Desire for solitude

– Depression

– Food craving (sweets)

– Speech disorders

  1. Aura: precedes hemicranias (pain on a hemicranium) by 5-60 minutes. It occurs in 20% of cases. May occur:

– Eye phenomena: sparkling scotoma, hemianopsia, amaurosis (vision loss).

– Paresthesia

– Vertigo

  1. Headache: the only constant element in 100% of cases, defining the migraine.

– Severe, with throbbing

– Most often unilateral

– Accompanied by nausea, vomiting, photophobia

– Aggravated by motion, light

– Determines the patient’s self-isolation in closed rooms

– Takes 2 -72 h

  1. Postdromal (postictal state):

– Sleepiness

– Tiredness

– Sometimes euphoria

– Can last up to 24 hours

Crises are separated by free intervals. A daily headache is not a migraine.

Diagnosis: on the basis of some criteria introduced by the International Headache Society. It is a diagnosis of exclusion, finally, in terms of a negative neurological exam! The following forms can be recognized:

  1. Migraine without aura: seizures lasting 4-72 hours, which meet at least two of the following conditions:

– Pain is unilateral

– Pain is pulsatory

– The intensity of the pain is moderated or severe

– The pain is aggravated by physical activity

– The pain is associated with one or more of the following:

  • nausea
  • vomiting
  • photophobia
  • phonophobia
  1. Migraine with aura: two or more seizures preceded by aura, symptoms of which should not take more than 60 minutes and completely reversible. Pain can take 4-72 hours and must meet at least two of the conditions described above.

Differential diagnosis of migraine

  1. associated headaches, secondary or symptomatic from trauma, vascular disturbances, metabolic septic, skull pathology, pathology of the eye, nose, throat, ears, sinuses, teeth, mouth.
  2. Migraine-like symptoms but with objective signs during the neurological exam:

-cerebral malformations-> angioma, aneurysms, carotid-cavernous fistula

– recurrent vascular acute cerebral-vascular insufficiency

-tumor of the lateral ventricles, third ventricle, occipital tumors

Migraine Treatment

  1. I) Using drugs – it is initiated only when the diagnostic is of certainty; therapy has many side effects and low efficiency (there is no ideal therapy for this).
  2. A) In the acute phase:
  3. Simple analgesics:

– Acetylsalicylic acid (Analgin) -> central mechanism (thalamic) and peripheral (COX irreversibly blocking> inhibiting PG) -> 2 g / 24 h

– Paracetamol (Panadol) -> 1-2 g / 24 h

– Metamizol (Algocalmin, Novalgin) -> 1-2 g / 24 h

  1. NSAIDs:

– Indomethacin

– Diclofenac

– Ibuprofen

– Naproxen

  1. Ergot derivatives: they couple with the receptors of 5HT, which mediate the neurogenic inflammation in the brain arterioles and have serotonergic action. Chronic use at high doses can lead to ergotism and may turn the migraine into a daily headache. They are augmented by caffeine, which increases the absorption and with which they mix.
  2. Selective 5HT1 receptor antagonists: sumatriptan (Imigran)
  3. B) Prevention: for crisis lasting more than 3 days per month, interrupting normal activity, with long-lasting attacks (over 72 hours) or extreme intensity, for which acute phase therapies do not work, have side effects or are contraindicated:
  4. Beta-blockers: propranolol, etc.
  5. Calcium channel blockers: verapamil, nifedipine
  6. 5HT2 antagonists: pizotifen (Sandomigran), methysergide (Deseril), lisuride (Lisenil), dimetotiazin (Migristen), oxetorone (Nocertone), etc.
  7. Antidepressants: imipramine, doxepin, amitriptyline.
  8. Tranquilizers: oxazepam, clorazepate (Tranxene), chlordiazepoxide, diazepam.
  9. Aspirin.
  10. Hormonal: estradiol.
  11. Magnesium preparations.
  12. Vitamin preparations.
  13. Antiemetics.
  14. II) Surgical: only in cases which are not responding to treatment. It aims to break the trigeminal afferents, arterial occlusion, disruption of vasodilator pathways, and disruption of the efferent pathways of pain.

III) Psychotherapy: fighting panic attacks, anxiety.

Posted in Brain

Traveler’s diarrhea – remedies


The most common cause for traveler’s diarrhea is the bacterium E. coli. This widespread organism normally lives in our intestines and plays a role in digestion. But different types of E. coli can cause diarrhea by producing a toxin that prevents the intestines to absorb water consumed as liquids and food.

diareea-calatorului-300x175Bacteria of the genus Shigella and Salmonella can cause diarrhea and a small number of cases are caused by rotaviruses or by the parasite called Giardia lamblia. Food changes, fatigue, jet lag and altitude sickness were all incriminated in motivating traveler’s diarrhea, but about half of the cases remain unexplained.

There are ways to help your body fight with traveler’s diarrhea

  1. Drink water. Through dehydration due to electrolyte and water loss, you could die. If you fail to replace the lost fluids, you could be dehydrated within only 24 hours.
  2. Check your urine. The darker the urine, the more fluids you need. Urine should be light, pale yellow. If dark urine persists, especially if the stool is light yellow, you must immediately do liver tests.
  3. Use a rehydration solution. These drinks contain sugar and salt and help replace electrolytes lost through diarrhea. They also help the intestines absorb water. Rehydration sachets are sold without prescription.
  4. Drink fruit juice or weak tea, sweetened. Concentrated bottled drinks are also very good, but shake them before drinking in order to remove the carbonic acid.
  5. An antidiarrheal drug containing pectin and bismuth basic carbonate is sold without prescription and can be an ally of the travelers. It gives consistency and firmness to the stools and kills bacteria. Do not worry if the tongue and diarrhea stool become black; it is a side effect of bismuth carbonate.
  6. Natural fiber-based laxatives for constipation also help in diarrhea. Some may absorb an amount of water representing about 60 times their weight, forming a gel in the intestine.
  7. Go for an exam. If the symptoms persist upon returning home, a coprologic exam will have to be done. Without treatment, bacteria and parasites can remain the unwanted holiday souvenirs for months.
Posted in Remedies

Heartburn – remedies for gastroesophageal heartburn


Heartburn (feeling discomfort behind the breastbone) is caused by a reflux of the acid from the stomach contents back into the esophagus.

Sometimes, the symptoms are so strong that the pain is felt like a heart attack. In some cases, it can create confusion because heart pain can be felt in the same area.

HeartburnOther accompanying symptoms of heartburn are: acid taste in the mouth, the pain gets worse while stooping or sleeping, nausea, bloating, nocturnal cough.

Overeating is the most common cause of the occurrence of reflux and heartburn. There are other causes, but they occur more rarely.

We suggest some remedies for heartburn

  • Do not overeat. When the stomach is full, it may lead to the reflux of the content into the esophagus. Typically, reflux occurs in people who eat fast or make excesses.
  • Excess acid occurs due to fatty and fried food. These foods stagnate in the stomach longer. Avoid fatty meats and dairy foods.
  • Often, it is believed that foods like chili peppers and spices are responsible for reflux, but they are not.
  • Citrus fruits like oranges and lemons are acidic, but the acid content in them has a different composition.
  • Drugs: there are antacids sold over the counter that neutralize the acid produced in the stomach. Take this medicine before or after meals.
  • Milk is a food that can lead to an improvement, but not really. Milk increases the production of stomach acid.
  • Mint has a relaxing effect of the lower esophageal sphincter (the one that prevents reflux). Avoid mint candy or teas.
  • Other foods that relax the esophageal sphincter are beer, wine, alcoholic drinks, tomatoes.
  • Caffeinated drinks have a negative effect on the already inflamed esophagus and appear to have a relaxing effect on the sphincter. Avoid coffee, tea, cola.
  • Do not drink hot beverages as they can worsen the injury in the esophagus. Drink the tea and coffee warm.
  • Do not consume chocolate: it has caffeine and a high level of fat (fat stimulates acid production).
  • Do not drink carbonated drinks that will expand the stomach and trigger the reflux.
  • Quit smoking.
  • Do not wear belts, skirts or too tight underwear because this stimulates reflux.
  • When you have to pick something on the floor, bend the knees. Otherwise it will compress the stomach and the acid will be forced to go up into the esophagus.
  • There are drugs that aggravate the heartburn such as antidepressants, sedatives, antibiotics, antihypertensives, analgesics, steroids.
  • Eat little and often and do not drink a lot of fluid while you’re at the table. Do not drink anything 2 hours before going to bed.
  • Try to avoid stress.

Do all these things and you will see an improvement in heartburn.

Posted in Remedies

Oral Herpes – remedies, tips

Oral Herpes

A significant percentage of the adult population have antibodies in their blood against Herpes Simplex Virus. In general, these people were exposed in childhood to the action of a highly contagious virus that causes herpes, which is very different from the herpes virus that causes genital herpes.

Oral HerpesNormally, after initial eruption the virus remains dormant in nerve cells, but occasionally it is reactivated. Before herpes reappears, you will find an unpleasant sensation of numbness, tingling, burning or itching on the lips or on the skin around the mouth.

There are several steps you can do to lessen the pain caused by herpes and to speed the healing

  • Most efficient medicine for herpes is an antiviral cream with the basic substance Acyclovir in a proportion of 5%. It is sold without prescription in pharmacies. Once symptoms appear, apply the cream on the area 5 times a day, 5 consecutive days. Treatment reduces the period of eruption and limits the pain. Acyclovir will take effect if given within 24 hours of the appearance of symptoms of tingling, preceding the actual herpes rash to appears.
  • If it is a herpes eruption, leave it alone. Keep clean and dry the herpes eruption.
  • Change your toothbrush. Toothbrush can accommodate herpes virus several days. Change your toothbrush when you notice that you are about to make a herpes. By doing so, you prevent multiple herpes eruptions. And once the wound has healed completely, replace your toothbrush again.
  • Do not hold your toothbrush in the bathroom. A wet toothbrush kept in a humid environment such as the bathroom, is a perfect environment for Herpes Simplex. Moisture helps to prolong life of viruses hosted by the toothbrush. Keep it in a dry place.
  • Use small tubes of toothpaste. The disease can be transmitted through toothpaste. Buy small tubes and replace them often.
  • Protect herpes with vaseline. You can protect a herpes rash covering it with a layer of grease. Use a small clean cotton swab.
  • Research shows that zinc applied to the first tingling sensation, help reduce the healing time. Try ointments available in pharmacies, with a content of 1% zinc sulfate.
  • Identify patterns. What happened in your personal life right before your last herpes eruption appeared? What do you know about earlier herpes eruption? If you discover when you are most prone to herpes eruption, take lysine supplements when you are most vulnerable. Between regular triggers of herpes are stress, menstruation, sunlight, fatigue, various minor ailments and a wide variety of foods.
  • Numb it. Most products sold without prescription contain a quantity of emollient that reduces cracks, dries crusts and an agent that numbs, such as phenol and camphor.
  • Protect your lips from the sun or wind exposure was cited by all experts as a mean to prevent herpes eruptions.
  • Avoid foods high in arginine. Herpes virus, for its metabolism, needs the essential amino acid called arginine. Give up the arginine-rich foods: chocolate, cola drinks, peas, grains, peanuts, jelly, chestnuts and beer.
  • Exercise. There is evidence that exercises help to strengthen the immune system. The stronger your immune system is, the body is able to fight with viruses.
Posted in Remedies

Dry Eyes – simple treatments

dry eyes

Simple treatments for dry eyes. Dry eyes occur when they do not produce enough tears to keep them moist. Eye dryness is a common problem. Half of people over 40 years are dealing with dry eyes in one form or another, whether as an intermittent or persistent problem. Dry eye is a component of aging.

dry eyesBlinking of eyes forms a triple film consisting of water, fat and mucus. Around age 40, the tear glands begin to idle, producing less soothing liquid to the eye. The problem is even more serious in postmenopausal women because of hormonal changes of the body which dry secretions, including tears.

The term “dry eye” is just a name for a complex and annoying condition characterized by redness, burning, itching, sensitivity to light and the desire to scratch your eyes.

Sun, wind, cold, central heating and air conditioning in homes and offices, a long time spent in front of the computer or TV and even high altitudes can cause later troubles if you have dry eyes.

A wide range of medications such as decongestants, antihistamines, diuretics, anesthetics, antidepressants, heart medications, remedies for ulcers, chemotherapy and drugs containing beta-blockers may reduce tear production and cause dry eyes.

What you can do for dry eyes

1. Apply a warm compress. If you occasionally feel dry eyes, put a warm compress on the eyelids for 5-10 minutes, 2 or 3 times a day. This stimulates the flow of tears.

2. Opt for an ointment. To combat dry eye cases where sleep is unbearable and for pain ease, when you go to bed, apply a mixture of eyewash (artificial tears) with ointment, which binds moisture in the tissues. These ointments sold over without prescription contain petrolatum and mineral oil and have the quality that adheres to tissue and do not leak. These ointments may blur your vision for a short time, so it’s best to apply them when you are in bed already.

3. Avoid contact lenses. If you feel your eyes dry and irritated, the last thing you need are contact lenses. In essence, contact lenses act like a sponge that absorb natural or artificial tears. When the eyes are already producing fewer tears, contact lenses can further dry eyes and irritate them, redden and make them sensitive to light.

4. Wear glasses with side protection. As the wind and sun can dry your eyes, wear sunglasses with extra protection.

5. A jet of heat or air conditioning may be just what the body needs to make more bearable traveling by car, but a direct and continuous flow of hot or cold air can irritate eyes.

6. Enjoy the fresh air. Open the window and let in fresh air. This way the air is moistened and can do much good for the eye.

7. Bring vegetation in the room. Buy an apartment plant or two and put them in the rooms where you spend the most time. They can grow room humidity.

8. Blink often. If occupational activity involves spending many hours at the computer, make occasional pauses to blink.

Posted in Eye, Remedies

Premenstrual Syndrome – Treatment


PMSA week before the menstrual cycle some women suffer from premenstrual syndrome. It consists of: tender and painful breasts, headaches, emotional instability. These manifestations can even disrupt normal life of the woman.

This syndrome occurs at a fairly large number of women (a third, even half) with an age between 20 and 50 years.

The cause of premenstrual syndrome is hormonal fluctuation that present women before the menstrual cycle (estrogen and progesterone hormones are responsible).

Other symptoms: mental instability, women are aggressive, exhausted and often appears depression, bloating, weight gain, skin disorders, appear cravings to consume increased amounts of sweets and alcohol.

Here are some remedies for premenstrual syndrome

  • Stress has a great influence on women with premenstrual syndrome. The advice is to stay in a room with soothing colors, to listen to calm music. These things can induce a state of calm that will definitely help.
  • No shallow breathing: breathe in deeply. Shallow breathing lowers the body’s energy (reduced oxygen intake). Pull air in the chest and exhale gently.
  • In menstrual syndrome it may occur muscle and blood circulation problems. A remedy in this aspect would be to have sex and if possible to reach orgasm. Sex accelerates blood circulation.
  • Take a warm bath in which to put a cup of sea salt and a sodium bicarbonate. Stay in the bath water for 20 minutes.
  • “Mielareaua” is an effective remedy for premenstrual syndrome manifestations. About half of women who used this remedy for 3 cycles observed improvements in symptoms. The plant can be taken as capsules, tincture or tea. Time of day to be taken is in the morning.
  • Insomnia also may be a manifestation of premenstrual syndrome. Before insomnia appears, with a few nights before try to go to bed a few hours faster, so your symptoms will occur when you will be ready.
  • Eat light and frequent meals, without large amounts of sugar and fat.
  • Eat a high amount of fibers. Fibers help to eliminate the excess of estrogen in the body. The fibers are found in vegetables, cereals, bread.
  • Caffeine should be consumed in small amounts: reduce coffee intake, tea, coca-cola. Caffeine increases breast tenderness, anxiety and irritability.
  • Avoid salt and alcohol.
Posted in Female Genital

Irritable Colon – simple remedies

irritable colon

irritable colonIrritable Colon Remedies. No etiology is known for irritable colon, but it is believed that intestinal muscles contract stronger and more prolonged in people who suffer from this condition.

The most common manifestations of this syndrome are diarrhea, constipation, abdominal pain, mucus in stool. It should be identified and removed from the diet the foods and drinks that cause these symptoms. Also, some events can trigger episodes of diarrhea and abdominal pain.

It should be noted that irritable colon syndrome is a functional disorder and is not affected any structure (colon). There is not a risk factor for colon cancer.

What you can do for irritable colon

  • Between irritable colon and stress is a connection. Do not let stress overwhelm you. If you begin to have abdominal pain, take a deep breath and calm yourself!
  • Relief of symptoms should occur when you do something that will relax. You can try relaxation techniques, meditation or yoga. Experiment and find what works in your case.
  • If you have irritable colon it means that your gut has an overreaction when you make changes regarding eating habits, stress and hormonal fluctuations. Try keeping a record time of two weeks of what will cause bowel irritation. You should identify a pattern.
  • Note the foods and drinks you consume. I will help you identify which of them will accentuate your irritable colon manifestations.
  • Your diet should contain a large amount of dietary fiber. This diet is successful for many people. The fibers have an effect on constipation and diarrhea. Eat bran, cereals, fruits and vegetables.
  • Drink about 6-8 glasses of water daily. Water helps the intestines to function better.
  • Do you have lactose intolerance? Do not drink milk and see what happens. Many who believe they have irritable colon they actually have a lactose intolerance.
  • Nicotine from cigarette worsens symptoms. Give up smoking.
  • Sorbitol contained in gum and sweets has potential of worsening the symptoms.
  • The contractions of the colon may be exacerbated by fats. Reduce fats, sauces, fried foods and oils.
  • Avoiding spices is also recommended.
  • Caffeine and coffee tars can cause discomfort for those with irritable colon. If you do not want to give up coffee, drink decaffeinated coffee.
  • One last tip would be to eat little and often.
Posted in Colon


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